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A '''choke-out''' is a [[hand-to-hand combat]] tactic involving the use of a [[chokehold]] to cause [[Syncope (medicine)|syncope]], or temporary loss of consciousness, at which point the choke is released. Common chokeholds in grappling used to accomplish a choke-out include the [[rear naked choke]], [[arm triangle]], [[triangle choke]], and the [[Guillotine choke|guillotine]].
A '''choke-out''' is a [[hand-to-hand combat]] tactic involving the use of a [[chokehold]] to cause [[Syncope (medicine)|syncope]], or temporary loss of consciousness, at which point the choke is released. Common chokeholds in grappling used to accomplish a choke-out include the [[rear naked choke]], [[arm triangle]], [[triangle choke]], and the [[Guillotine choke|guillotine]].


The mechanics behind choke-outs are disputed. It has been explained as resulting from directly constraining blood flow to the [[brain]]. A competing theory involves compression of the [[baroreceptor]]s of the [[carotid artery]], confusing the body into thinking [[blood pressure]] has risen. Due to the [[baroreflex]], this causes [[vasodilation]], or widening of the brain's blood vessels intended to relieve high pressure. Since no blood pressure increase has actually occurred, the dilation causes a dramatic decrease in blood pressure to the brain, or [[brain ischemia]], which then causes loss of consciousness.
The mechanism can be explained as resulting from directly constraining blood flow to the [[brain]] through constriction of the carotid arteries. An additional mechanism involves compression of the [[baroreceptor]]s of the [[carotid arteries]], confusing the body into thinking [[blood pressure]] has risen. Due to the [[baroreflex]], this inhibits sympathetic vasomotor and cardiac stimulation and increases parasympathetic stimulation of the heart causing [[vasodilation]] and a lowered heart rate.<ref name="sciencedirect-baroreflex">{{Cite web |last=Gropper |first=Michael A. |title=Baroreflex |url=https://www.sciencedirect.com/topics/neuroscience/baroreflex |website=[[Science Direct]]}}</ref> This causes a dramatic decrease in blood flow especially to regions above the heart (e.g. the brain) due the need of a high pressure to flow against gravity which in turn results in less blood flow to the brain ([[brain ischemia]]), which then causes loss of consciousness.<ref name="sciencedirect-baroreflex" /> These explanations, however, do not exclude each other but are 2 components explaining less blood flow to the brain: 1) constriction of carotid arteries leading blood towards the brain directly causing less blood flow to the brain 2) lower systemic blood pressure (thereby also decreasing blood flow to the brain through the vertebral arteries - these being the only alternative blood supply to the brain<ref>{{cite web |url=https://teachmeanatomy.info/neuroanatomy/vessels/arterial-supply/ |title=The Arterial Supply to the Central Nervous System |website=Teach me Anatomy}}</ref>).


Choke-outs should not be confused with [[erotic asphyxiation]] or the [[fainting game]], wherein a person loses consciousness intentionally in order to experience a particular sensation. A choke-out should also not be confused with medical conditions that cause fainting without the application of a chokehold.
Choke-outs should not be confused with [[erotic asphyxiation]] or the [[fainting game]], wherein a person loses consciousness intentionally in order to experience a particular sensation. A choke-out should also not be confused with medical conditions that cause fainting without the application of a chokehold.
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[[Chokehold]]s can be divided into two primary categories: "[[Chokehold#Blood choke|blood choke]]s" and "air chokes". A blood choke disrupts blood circulation to the brain, while an air choke disrupts breathing. Blood chokes can be applied to efficiently cause loss of consciousness, i.e. a choke-out, while air chokes do not usually cause loss of consciousness without prolonged application (though air chokes are used to cause discomfort).
[[Chokehold]]s can be divided into two primary categories: "[[Chokehold#Blood choke|blood choke]]s" and "air chokes". A blood choke disrupts blood circulation to the brain, while an air choke disrupts breathing. Blood chokes can be applied to efficiently cause loss of consciousness, i.e. a choke-out, while air chokes do not usually cause loss of consciousness without prolonged application (though air chokes are used to cause discomfort).


Blood chokes constrain or disrupt blood circulation to the brain. This is accomplished by compressing one or both of the [[carotid arteries]] and/or the [[jugular vein]]s, ideally with little to no pressure applied to the airway.
Blood chokes constrain or disrupt blood circulation to the brain. This is accomplished by compressing one or both of the [[carotid arteries]] and/or the [[jugular vein]]s, ideally with little to no pressure applied to the airway. However, it is very rare and difficult to accomplish by compressing only one side.


One theory on the mechanics of choke-outs claims that unconsciousness results from the direct constraint of blood flow to the brain, causing [[cerebral hypoxia]].<ref>{{cite journal |last1=Mitchell |first1=Jamie R. |last2=Roach |first2=Dan E. |last3=Tyberg |first3=John V. |last4=Belenkie |first4=Israel |last5=Sheldon |first5=Robert S. |title=Mechanism of loss of consciousness during vascular neck restraint |journal=Journal of Applied Physiology |volume=112 |issue=3 |pages=396–402 |year=2012 |pmid=22096121 |doi=10.1152/japplphysiol.00592.2011 |lay-url=http://www.bloodyelbow.com/2012/4/6/2929371/university-of-calgary-rear-naked-choke-study-VNR-police-research-explains |lay-source=[[SB Nation]] |lay-date=April 6, 2012 }}</ref>
Unconsciousness results mainly from the direct constraint of blood flow to the brain, causing [[cerebral hypoxia]].<ref>{{cite journal |last1=Mitchell |first1=Jamie R. |last2=Roach |first2=Dan E. |last3=Tyberg |first3=John V. |last4=Belenkie |first4=Israel |last5=Sheldon |first5=Robert S. |title=Mechanism of loss of consciousness during vascular neck restraint |journal=Journal of Applied Physiology |volume=112 |issue=3 |pages=396–402 |year=2012 |pmid=22096121 |doi=10.1152/japplphysiol.00592.2011}}
*{{cite web |author=Ben Thapa |date=April 6, 2012 |title=How Do Rear Naked Chokes Work? University of Calgary Study Explains |website=[[SB Nation]] |url=http://www.bloodyelbow.com/2012/4/6/2929371/university-of-calgary-rear-naked-choke-study-VNR-police-research-explains}}</ref>


An additional explanation is seen in the introduction and is caused by the baroreceptor reflex. This explains why fainting can also happen with just a very short application of force as this can cause systemic blood pressure to drop dramatically in some people thereby lowering the blood flow to the brain even after release of the constrictive force on the carotid arteries.<ref name="sciencedirect-baroreflex" />
{{Citation needed span|text=Another more complex theory states that as pressure is applied to the carotid artery, [[baroreceptors]] (the body's natural blood pressure monitors) within the artery are also compressed. When they sense too much pressure in an artery that feeds the brain (such as those in the neck), they signal the brain to [[vasodilation|dilate]] (widen) its blood vessels in order to relieve pressure ([[baroreflex]] causing [[vasodilation]]). Since blood pressure has not actually increased as the brain thinks it has, the dilation causes pressure to drop dramatically, and can be sufficient to cause cerebral hypoxia.|date=November 2012}}


== Dangers ==
== Dangers ==
There is debate over the dangers of choke-outs. After 4 to 6 minutes of sustained [[cerebral anoxia]], permanent [[brain damage]] will begin to occur,<ref name=source4>{{MedlinePlusEncyclopedia|001435|Cerebral hypoxia}}</ref> but the long-term effects of a controlled choke-out for less than 4 minutes (as most are applied for mere seconds and released when unconsciousness is achieved) are disputed.<ref>{{cite journal |first1=Ralph E. |last1=Cash |date=November 2007 |title=A Dangerous High |journal=Principal Leadership |volume=8 |issue=3 |pages=10–3 |url=http://www.nasponline.org/resources/principals/The%20Choking%20Game-NASSP%20Nov%2007.pdf}}</ref> There is always risk of short-term memory loss, hemorrhage and harm to the retina, concussions from falling when unconscious, stroke, seizures, permanent brain damage, coma, and even death.<ref>{{cite journal |last1=Neumann-Potash |first1=L. |date=Fall 2006 |title=The choking game |journal=California Pediatrician |issn=0882-3421 |pages=22 }}</ref>
The long-term effects of a controlled choke-out for less than 4 minutes (as most are applied for mere seconds and released when unconsciousness is achieved) are disputed,<ref>{{cite journal |first1=Ralph E. |last1=Cash |date=November 2007 |title=A Dangerous High |journal=Principal Leadership |volume=8 |issue=3 |pages=10–3 |url=http://www.nasponline.org/resources/principals/The%20Choking%20Game-NASSP%20Nov%2007.pdf}}</ref> but the 5 minute mark is largely considered unsafe.<ref name=source4>{{MedlinePlusEncyclopedia|001435|Cerebral hypoxia}}</ref> There is always risk of short-term memory loss, hemorrhage and harm to the retina, concussions from falling when unconscious, stroke, seizures, permanent brain damage, coma, and even death.<ref>{{cite journal |last1=Neumann-Potash |first1=L. |date=Fall 2006 |title=The choking game |journal=California Pediatrician |issn=0882-3421 |pages=22 }}</ref>


Some argue that when pressure is applied to the carotid artery, the baroreceptors send a signal to the brain via the [[glossopharyngeal nerve]]<ref>CONSTANTINOS H. DAVOS, LEWIS CERI DAVIES, MASSIMO PIEPOLI. The Effect of Baroreceptor Activity on Cardiovascular Regulation. Hellenic J Cardiol 43: 145-155, 2002. Retrieved from {{cite web |url=http://hellenicjcardiol.com/archive/full_text/2002/5/2002_5_145.pdf |title=Archived copy |accessdate=2015-11-23 |url-status=dead |archive-url=https://web.archive.org/web/20151123035956/http://www.hellenicjcardiol.com/archive/full_text/2002/5/2002_5_145.pdf |archive-date=2015-11-23 }}</ref> and the [[heart]] via the [[vagus nerve]]. This signal tells the heart to reduce volume of blood per heartbeat, typically up to one-third, in order to further relieve high pressure. There is a slight chance of the rate dropping to zero, or [[flatline]] ([[asystole]]).<ref name=source5>{{cite web |first=Jay |last=Wiseman |year=1997 |title=The Medical Realities Of Breath Control Play |url=http://www.leathernroses.com/generalbdsm/wisemanbreathcontrol1.htm}}{{self-published inline|date=August 2015}}</ref> However, there are several studies that showed choking out will result in a few seconds of flat line ECG for a few seconds at least in half of the subjects.<ref name=pmid8053660>{{cite journal |last1=Lempert |first1=T. |last2=Bauer |first2=M. |last3=Schmidt |first3=D. |title=Syncope: A videometric analysis of 56 episodes of transient cerebral hypoxia |journal=Annals of Neurology |volume=36 |issue=2 |pages=233–7 |year=1994 |pmid=8053660 |doi=10.1002/ana.410360217 }}</ref> This might suggest that choking out or syncope is not as safe as it was assumed to be previously.
Some argue that when pressure is applied to the carotid artery, the baroreceptors send a signal to the brain via the [[glossopharyngeal nerve]]<ref>{{Cite journal |last=Davos |first=Constantinos |last2=Davies |first2=Lewis Ceri |last3=Piepoli |first3=Massimo |date=2002 |title=The effect of baroreceptor activity on cardiovascular regulation. |url=https://hellenicjcardiol.org/archive/full_text/2002/5/2002_5_145.pdf |url-status=live |journal=Hellenic J Cardiol |volume=43 |pages=143–155 |archive-url=https://web.archive.org/web/20210616145143/https://hellenicjcardiol.org/archive/full_text/2002/5/2002_5_145.pdf |archive-date=16 June 2021 |access-date=23 March 2022}}</ref> and the [[heart]] via the [[vagus nerve]]. This signal tells the heart to reduce volume of blood per heartbeat, typically up to one-third, in order to further relieve high pressure. There is a slight chance of the rate dropping to zero, or [[flatline]] ([[asystole]]).<ref name=source5>{{cite web |first=Jay |last=Wiseman |year=1997 |title=The Medical Realities Of Breath Control Play |url=http://www.leathernroses.com/generalbdsm/wisemanbreathcontrol1.htm}}</ref>{{self-published inline|date=August 2015}} However, there are several studies that showed choking out will result in a few seconds of flat line ECG for a few seconds at least in half of the subjects.<ref name=pmid8053660>{{cite journal |last1=Lempert |first1=T. |last2=Bauer |first2=M. |last3=Schmidt |first3=D. |title=Syncope: A videometric analysis of 56 episodes of transient cerebral hypoxia |journal=Annals of Neurology |volume=36 |issue=2 |pages=233–7 |year=1994 |pmid=8053660 |doi=10.1002/ana.410360217 }}</ref> This might suggest that choking out or syncope is not as safe as it was assumed to be previously.


Some argue that with thousands of tournaments since the sport of [[Judo]] began in 1882, hundreds of thousands of chokes have been applied, and the probability of hundreds if not thousands of choke-outs, with no reported deaths due to chokes, the chances of asystole are slim.<ref name=source6>{{cite journal |last1=Koiwai |first1=E. Karl |title=Deaths allegedly caused by the use of 'choke holds' (shime-waza) |journal=Journal of Forensic Sciences |volume=32 |issue=2 |pages=419–32 |year=1987 |pmid=3572335 |url=http://judoinfo.com/chokes6.htm }}</ref> It might be true that no direct death has reported as a result of chokes but there are numerous reports of these chokes turned out to strokes leaving the subject with permanent brain damage or possible more elaborated long-term effects <ref name=source17>{{cite journal |last1=Urkin |first1=Jacob |last2=Merrick |first2=Joav |title=The choking game or suffocation roulette in adolescence |journal=International Journal of Adolescent Medicine and Health |volume=18 |issue=2 |pages=207–8 |year=2006 |pmid=16894858 |doi=10.1515/IJAMH.2006.18.2.207 }}</ref>
Some argue that with thousands of tournaments since the sport of [[Judo]] began in 1882, hundreds of thousands of chokes have been applied, and the probability of hundreds if not thousands of choke-outs, with no reported deaths due to chokes, the chances of asystole are slim.<ref name=source6>{{cite journal |last1=Koiwai |first1=E. Karl |title=Deaths allegedly caused by the use of 'choke holds' (shime-waza) |journal=Journal of Forensic Sciences |volume=32 |issue=2 |pages=419–32 |year=1987 |pmid=3572335 |url=http://judoinfo.com/chokes6.htm }}</ref> It might be true that no direct deaths have been reported as a result of chokes, but there are numerous reports of these chokes turning out to be strokes, leaving the subject with permanent brain damage or possibly more elaborate, long-term effects <ref name=source17>{{cite journal |last1=Urkin |first1=Jacob |last2=Merrick |first2=Joav |title=The choking game or suffocation roulette in adolescence |journal=International Journal of Adolescent Medicine and Health |volume=18 |issue=2 |pages=207–8 |year=2006 |pmid=16894858 |doi=10.1515/IJAMH.2006.18.2.207 }}</ref>


== Effects ==
== Effects ==
The [[American Neurological Association]]'s study entitled “Syncope: A videometric analysis of 56 episodes of transient cerebral hypoxia” observed the effects of cerebral hypoxia on 42 test subjects who completely lost consciousness. Their syncope state lasted 12.1 seconds, plus or minus 4.4 seconds. Muscle jerks occurred in 90% of patients. The most common pattern of movement consisted of multifocal arrhythmic jerks (uncoordinated [[spasm]]s of multiple muscle groups) in both proximal and distal muscles. Additional movements also occurred such as: righting movements (if the patient had slumped one way while falling asleep they woke up and immediately corrected, if not overcorrected), oral automatisms, and head turns. In most of the patients their eyes remained open. Sixty percent of the patients reported having visual and auditory hallucinations.<ref name=pmid8053660/>
The [[American Neurological Association]]'s study entitled "Syncope: A videometric analysis of 56 episodes of transient cerebral hypoxia" observed the effects of cerebral hypoxia on 42 test subjects who completely lost consciousness. Their syncope state lasted 12.1 seconds, plus or minus 4.4 seconds. Muscle jerks occurred in 90% of patients. The most common pattern of movement consisted of multifocal arrhythmic jerks (uncoordinated [[spasm]]s of multiple muscle groups) in both proximal and distal muscles. Additional movements also occurred such as: righting movements (if the patient had slumped one way while falling asleep they woke up and immediately corrected, if not overcorrected), oral automatisms, and head turns. In most of the patients their eyes remained open. Sixty percent of the patients reported having visual and auditory hallucinations.<ref name=pmid8053660/>


== References ==
== References ==

Latest revision as of 15:24, 1 March 2023

A choke-out is a hand-to-hand combat tactic involving the use of a chokehold to cause syncope, or temporary loss of consciousness, at which point the choke is released. Common chokeholds in grappling used to accomplish a choke-out include the rear naked choke, arm triangle, triangle choke, and the guillotine.

The mechanism can be explained as resulting from directly constraining blood flow to the brain through constriction of the carotid arteries. An additional mechanism involves compression of the baroreceptors of the carotid arteries, confusing the body into thinking blood pressure has risen. Due to the baroreflex, this inhibits sympathetic vasomotor and cardiac stimulation and increases parasympathetic stimulation of the heart causing vasodilation and a lowered heart rate.[1] This causes a dramatic decrease in blood flow especially to regions above the heart (e.g. the brain) due the need of a high pressure to flow against gravity which in turn results in less blood flow to the brain (brain ischemia), which then causes loss of consciousness.[1] These explanations, however, do not exclude each other but are 2 components explaining less blood flow to the brain: 1) constriction of carotid arteries leading blood towards the brain directly causing less blood flow to the brain 2) lower systemic blood pressure (thereby also decreasing blood flow to the brain through the vertebral arteries - these being the only alternative blood supply to the brain[2]).

Choke-outs should not be confused with erotic asphyxiation or the fainting game, wherein a person loses consciousness intentionally in order to experience a particular sensation. A choke-out should also not be confused with medical conditions that cause fainting without the application of a chokehold.

Mechanics[edit]

The vulnerable carotid artery, (large, red tube), and the vagus nerve running parallel on its left

Chokeholds can be divided into two primary categories: "blood chokes" and "air chokes". A blood choke disrupts blood circulation to the brain, while an air choke disrupts breathing. Blood chokes can be applied to efficiently cause loss of consciousness, i.e. a choke-out, while air chokes do not usually cause loss of consciousness without prolonged application (though air chokes are used to cause discomfort).

Blood chokes constrain or disrupt blood circulation to the brain. This is accomplished by compressing one or both of the carotid arteries and/or the jugular veins, ideally with little to no pressure applied to the airway. However, it is very rare and difficult to accomplish by compressing only one side.

Unconsciousness results mainly from the direct constraint of blood flow to the brain, causing cerebral hypoxia.[3]

An additional explanation is seen in the introduction and is caused by the baroreceptor reflex. This explains why fainting can also happen with just a very short application of force as this can cause systemic blood pressure to drop dramatically in some people thereby lowering the blood flow to the brain even after release of the constrictive force on the carotid arteries.[1]

Dangers[edit]

The long-term effects of a controlled choke-out for less than 4 minutes (as most are applied for mere seconds and released when unconsciousness is achieved) are disputed,[4] but the 5 minute mark is largely considered unsafe.[5] There is always risk of short-term memory loss, hemorrhage and harm to the retina, concussions from falling when unconscious, stroke, seizures, permanent brain damage, coma, and even death.[6]

Some argue that when pressure is applied to the carotid artery, the baroreceptors send a signal to the brain via the glossopharyngeal nerve[7] and the heart via the vagus nerve. This signal tells the heart to reduce volume of blood per heartbeat, typically up to one-third, in order to further relieve high pressure. There is a slight chance of the rate dropping to zero, or flatline (asystole).[8][self-published source?] However, there are several studies that showed choking out will result in a few seconds of flat line ECG for a few seconds at least in half of the subjects.[9] This might suggest that choking out or syncope is not as safe as it was assumed to be previously.

Some argue that with thousands of tournaments since the sport of Judo began in 1882, hundreds of thousands of chokes have been applied, and the probability of hundreds if not thousands of choke-outs, with no reported deaths due to chokes, the chances of asystole are slim.[10] It might be true that no direct deaths have been reported as a result of chokes, but there are numerous reports of these chokes turning out to be strokes, leaving the subject with permanent brain damage or possibly more elaborate, long-term effects [11]

Effects[edit]

The American Neurological Association's study entitled "Syncope: A videometric analysis of 56 episodes of transient cerebral hypoxia" observed the effects of cerebral hypoxia on 42 test subjects who completely lost consciousness. Their syncope state lasted 12.1 seconds, plus or minus 4.4 seconds. Muscle jerks occurred in 90% of patients. The most common pattern of movement consisted of multifocal arrhythmic jerks (uncoordinated spasms of multiple muscle groups) in both proximal and distal muscles. Additional movements also occurred such as: righting movements (if the patient had slumped one way while falling asleep they woke up and immediately corrected, if not overcorrected), oral automatisms, and head turns. In most of the patients their eyes remained open. Sixty percent of the patients reported having visual and auditory hallucinations.[9]

References[edit]

  1. ^ a b c Gropper, Michael A. "Baroreflex". Science Direct.
  2. ^ "The Arterial Supply to the Central Nervous System". Teach me Anatomy.
  3. ^ Mitchell, Jamie R.; Roach, Dan E.; Tyberg, John V.; Belenkie, Israel; Sheldon, Robert S. (2012). "Mechanism of loss of consciousness during vascular neck restraint". Journal of Applied Physiology. 112 (3): 396–402. doi:10.1152/japplphysiol.00592.2011. PMID 22096121.
  4. ^ Cash, Ralph E. (November 2007). "A Dangerous High" (PDF). Principal Leadership. 8 (3): 10–3.
  5. ^ MedlinePlus Encyclopedia: Cerebral hypoxia
  6. ^ Neumann-Potash, L. (Fall 2006). "The choking game". California Pediatrician: 22. ISSN 0882-3421.
  7. ^ Davos, Constantinos; Davies, Lewis Ceri; Piepoli, Massimo (2002). "The effect of baroreceptor activity on cardiovascular regulation" (PDF). Hellenic J Cardiol. 43: 143–155. Archived (PDF) from the original on 16 June 2021. Retrieved 23 March 2022.
  8. ^ Wiseman, Jay (1997). "The Medical Realities Of Breath Control Play".
  9. ^ a b Lempert, T.; Bauer, M.; Schmidt, D. (1994). "Syncope: A videometric analysis of 56 episodes of transient cerebral hypoxia". Annals of Neurology. 36 (2): 233–7. doi:10.1002/ana.410360217. PMID 8053660.
  10. ^ Koiwai, E. Karl (1987). "Deaths allegedly caused by the use of 'choke holds' (shime-waza)". Journal of Forensic Sciences. 32 (2): 419–32. PMID 3572335.
  11. ^ Urkin, Jacob; Merrick, Joav (2006). "The choking game or suffocation roulette in adolescence". International Journal of Adolescent Medicine and Health. 18 (2): 207–8. doi:10.1515/IJAMH.2006.18.2.207. PMID 16894858.

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