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Summary
| Description |
English: Rowen et al.'s proposal for how misprocessing of amyloid precursor protein (APP) leads to decreased long-term potentiation (LTP) and early and late cognitive decline in individuals with Alzheimer disease. From the article:
Rowan MJ, Klyubin I, Cullen WK, Anwyl R (April 2003). "Synaptic plasticity in animal models of early Alzheimer's disease". Philosophical transactions of the Royal Society of London. Series B, Biological sciences 358 (1432): 821–8. doi:10.1098/rstb.2002.1240. PMID 12740129. PMC: 1693153, http://journals.royalsociety.org/openurl.asp?genre=article&issn=0962-8436&volume=358&issue=1432&spage=821. |
| Date | |
| Source | Own work |
| Author | Diberri |
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I, the copyright holder of this work, release this work into the public domain. This applies worldwide. In some countries this may not be legally possible; if so: I grant anyone the right to use this work for any purpose, without any conditions, unless such conditions are required by law. |
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| Date/Time | Thumbnail | Dimensions | User | Comment | |
|---|---|---|---|---|---|
| current | 14:52, 9 November 2008 | | 239 × 322 (23 KB) | Diberri | {{Information |Description={{en|1=Rowen ''et al'''s proposal for how misprocessing of amyloid precursor protein (APP) leads to decreased long-term potentiation (LTP) and early and late cognitive decline in individuals with Alzheimer disease. From the arti |
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