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Summary
| Description |
English: Binding of CAPON results in a reduction of NMDA receptor/nitric oxide synthase (NOS) complexes, leading to decreased NMDA receptor–gated calcium influx and a catalytically inactive nitric oxide synthase. Overexpression of either the full-length or the novel shortened CAPON isoform as reported by Brzustowicz and colleagues is, therefore, predicted to lead to impaired NMDA receptor–mediated glutamate neurotransmission. |
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| Date | |||
| Source | Does the CAPON Gene Confer Susceptibility to Schizophrenia? | ||
| Author | Bang Wong | ||
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| Date/Time | Thumbnail | Dimensions | User | Comment | |
|---|---|---|---|---|---|
| current | 23:35, 2 January 2022 | | 1,370 × 956 (428 KB) | Jul059 | Lossless file size reduction |
| 11:34, 25 April 2009 |  | 1,370 × 956 (493 KB) | CopperKettle | {{Information |Description={{en|1=Binding of CAPON results in a reduction of NMDA receptor/nitric oxide synthase (NOS) complexes, leading to decreased NMDA receptor–gated calcium influx and a catalytically inactive nitric oxide synthase. Overexpression |
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